Many dogs are carriers for the DNA mutation but do not have the disease, while other dogs are affected and can have various eye problems as a result.
The damage to an individual’s vision depends on the severity of the syndrome. The problem in CEA is that an area of the retina (the nerve layer of the eye with rods and cones) and/or the choroid (the choroid is the blood vessel layer under the retina) does not develop the way it should because of DNA mutations. Eye defects seen in the syndrome have been seen in atypical breeds and mixed breeds with collie-type heritage as well. Generally spCeaking, most dogs with CEA have abnormal vision and their vision is affected by choroidal hypoplasia and colobomas (see below) in addition to the retinal detachments.Īffected dog breeds generally include collies, Australian shepherds, border collies, Shetland sheepdog, and the Lancashire heeler. The syndrome has no cure, although there are some treatments for some of the eye problems, such as surgery for progressive retinal detachments if they worsen over time. Thanks to eye examinations and selective breeding, the number of affected dogs has decreased significantly. Lowering that percentage has been difficult because CEA affected such a large percentage of the breed. Some retinal detachments can be progressive and worsen over time, so it can be a progressive disease.Ībout 50 years ago, veterinarians estimated that about 90 percent of collies carry the DNA mutation although not all those dogs have the syndrome itself. Symptoms of CEA, such as retinal folds, can appear after birth at a certain age and then disappear as the dog ages. The dogs who are destined to have the syndrome are born with the inherited DNA mutation, although the abnormalities may or may not be present at birth, so technically it is not a birth defect. The disease occurs in both eyes, but each eye may be affected differently.
Collie eye anomaly (CEA) is an inherited syndrome of eye problems that can cause vision defects.
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